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Further adding Magnesium, Apoaequorin and Coconut Oil as detailed in the August 2012 update section below; and NADH as detailed in the 2016 update below that.
Note: Often good results can be obtained from just the use of one supplement, so if there is a problem with patient compliance or other administration problems, or finance problems; do not give up, do what you can with your selected supplement(s) of choice. Our role is to provide you with all the main tools (nutrients) that can be helpful. Incrementally introducing them all over a period of time stands the best chance of complete success. Note: if finance is a problem then using 5 grams of MSM powder daily in juice or water will remove mercury from the brain, but nutritional support for detoxification such as Vitamin C and preferably also some Zell Immunocomplex will often be needed.
| August 2012 - Update
The most cutting-edge research suggests that Alzheimer's may stem from an inability of the brain to process glucose, leading to cell death. But the brain has another source of fuel: ketones, produced by the body from mid-chain triglycerides like those found in coconut oil. In fact, the most promising experimental Alzheimer's drugs being tested today is nothing more than medium-chain triglycerides derived from coconut oil. No need for drugs, for some people, coconut oil has proven to slow the progression of Alzheimer's and may have even prevented it. A good degree of return to normal occurs in some cases. One of those people is Steve Newport, whose Alzheimer's has slowed considerably. Some of his symptoms even reversed, thanks to the unlikely treatment prescribed by his wife, Dr. Mary Newport, a physician who runs a neonatology ward at a Tampa, Fla., hospital. Dr. Newport became determined to help her husband after the severity of his disease was discovered through an Alzheimer's test in which the person being tested is asked to draw a picture of a clock.
"So that was very, very devastating news," she said.
Dr. Newport began learning everything she could about her husband's disease.
"It appears to be a type of diabetes of the brain and it's a process that starts happening at least 10 or 20 years before you start having symptoms and it's very similar to type 1 or type 2 diabetes in that you develop a problem with insulin," she explained.
In this case, insulin problems prevent brain cells from accepting glucose, their primary fuel. Without it, they eventually die. But there is an alternative fuel -- ketones, which cells easily accept. Ketones are metabolised in the liver after you eat medium chain triglycerides, found in coconut oil. Dr. Newport added coconut oil to her husband Steve's diet. Just two weeks later, he took the clock test again and demonstrated stunning improvement.
"I thought at the time, was it just good luck? Was it a lot of prayer? Was it the coconut oil?" she said. "And I thought, well, we're going to keep the coconut oil going."
Three weeks later, Steve took the clock test a third time and continued to perform better on it. And it wasn't just intellectually, he also improved emotionally and physically.
"He was not able to run. He was able to run again," she recalled. "He could not read for about a year and a half, but after two or three months he was able to read."
"Instead of being very sluggish, not talking very much in the morning, he would come out in the morning with energy, talkative, and joking, and he could find his water and his utensils," Dr. Newport said.
She documented Steve's success in a book titled, Alzheimer's Disease: What If There Was A Cure? Dr. Newport received many "thank you" letters from people whose loved ones were suffering from Alzheimer's. Each of the letters claimed their family member was helped after they followed Steve's diet.
So until a high potency ketone ester is available to the general public, coconut oil is still a good ketone source. Just make sure it's pure - in other words, non-hydrogenated. Avoid any hydrogenated oil, including coconut oil, because hydrogenated oils are the same thing as dangerous trans fats. Check the list of ingredients for the word, "hydrogenated."
Some people are afraid to eat coconut oil because they think it's bad for your heart but it's actually very healthy. Dr. Beverly Teter is a lipid biochemist researcher at the University of Maryland and an expert in the area of dietary fat. She said that years ago, coconut oil was criticised for raising cholesterol. But scientists have since learned there are two kinds of cholesterol -- LDL, the bad kind, and HDL, which is very good for you. HDL, the good cholesterol, is the kind that coconut oil raises.
"So they put out the message that it increased serum cholesterol," Teter explained. "But the truth of the matter is, it was helping the profile of the serum cholesterol."
"That never has been corrected in the public press, and I think that's the reason people have misconceptions about it," she said.
Defence against Disease
Not only does coconut oil improve cholesterol levels, Teter said the way it helps the brains of some Alzheimer's patients can even be extended to people with Parkinson's disease, ALS, epilepsy, dementia, even schizophrenia and autism.
Our brains need a certain amount of calcium for good cell-to-cell communications. But as we age, our ability to get rid of excess calcium slows down. The brain's delicate calcium balance gets thrown out of whack, inflammation sets in and cell death skyrockets. Those inflamed and dying brain cells can trigger memory loss and even Alzheimer's. Recovery from Alzhimers can occur rapidly with some people by removing excess calcium according to this source: click here
The Importance of Magnesium
Whatever the original cause, the root of the problem of excess calcium in the brains nerve cells is most likely due to magnesium deficiency. Taking supplemental magnesium will have multiple benefits for the brain and the overall health. It promotes proper nerve function and reduces inflammation, a cause of nerve cell impairment and eventually destruction. It protects the brain from excess calcium and has even been found to reduce aluminium and mercury uptake of the brain, two major toxicities linked to nerve damage and hence Alzheimer’s.
November 2016 - Update
NADH (Nicotinamide adenine dinucleotide - Reduced form) is the most important Coenzyme (also known, appropriately, as Coenzyme 1) in releasing energy in every cell of the body. The final stage of energy production creates a substance known as ATP. ATP easily and readily releases energy everywhere in the body where it is needed for all functions of the body, this includes energy to think, make hormones and activate muscles and so on.
As we age and/or suffer from below par health or actual chronic disease we get locked into a downward spiral where a reduction in the ability of the cells to produce ATP occurs and that reduction causes more disease and poor health.
NADH provides electrical energy via electron (attached to hydrogen) donation. It can therefore be regarded as donating active hydrogen directly into the cells mitochondria. This is the most important facilitator of the aforementioned process in producing ATP. This process also helps to support the utilisation of oxygen AND is said to have the most powerful anti-oxidant effect in the body. These aspects make NADH a prime remedy for health, longevity and freedom from chronic disease.
Of course NADH is not the complete answer to health optimisation. Other nutrients found in foods, Superfoods and nutritional supplements are also needed. Other nutrients are involved in producing ATP in a complex process known as the Krebs cycle. However NADH is the highest energy provider in that cycle and even in cases of other enzymes lacking (for example co-enzyme Q10) NADH will help to produce ATP.
A broad mixture of nutrients and enzymes as found in specialised yeast cells in Zell Immunocomplex also stimulate ATP and will work synergistically with NADH to further enhance the bodies core energy production. Use of both would most likely necessitate reducing the initial doses, as per the general rules of not increasing the bodies energies too fast (increase the dose over a few weeks). However this may not apply to those with very low energy such as the elderly with moderate to severe Parkinson's or Alzheimer's where the more help to wake up the Krebs cycle the better.
NADH should take its rightful place as a potent support to any regenerative health regime alongside mineral and trace element supplementation, vitamins C, D; and where needed vitamin B12, further adding in Superfoods to provide a broad spectrum of phytonutrients.
Demarin, V. et al.: Treatment of Alzheimer‘s disease with stabilized oral nicotinamide adenine dinucleotide: a randomized, double-blind study. Drugs Exp Clin Res. 2004;30(1):27-33.
This study was designed to evaluate the effect of stabilized oral reduced nicotinamide adenine dinucleotide (NADH) on cognitive functioning in patients with Alzheimer‘s disease (AD). NADH is a coenzyme that plays a key role in cellular energy production and stimulates dopamine production. In previous trials NADH has been shown to improve cognitive functioning in patients with Parkinson‘s disease, depression and AD. The present trial was a randomized, placebo- controlled, matched-pairs, double-blind, 6-month clinical study. Patients with probable AD (n = 26) were randomized to receive either stabilized oral NADH (10 mg/day) or placebo. Twelve pairs of subjects were matched for age and baseline total score on the Mattis Dementia Rating Scale (MDRS) and the Mini Mental State Examination.
After 6 months of treatment, subjects treated with NADH showed no evidence of progressive cognitive deterioration and had significantly higher total scores on the MDRS compared with subjects treated with placebo (p < 0.05). Analysis of MDRS subscales revealed significantly better performance by NADH subjects on measures of verbal fluency (p = 0.019), visual-constructional ability (p = 0.038) and a trend (p = 0.08) to better performance on a measure of abstract verbal reasoning. There were no differences between groups in measures of attention, memory, or in clinician ratings of dementia severity (Clinical Dementia Rating). Consistent with earlier studies, the present findings support NADH as a treatment for Alzheimer‘s disease.
Another study on those with Alzheimer's undertaken by Georgetown University medical center (approved trial by FDA) found that 10 mg daily of NADH resulted in 100% halt in the progress of the disease over a 6 month period. There where also significant improvements in mental functioning as ascertained by standard assessment tests for Alzheimer's.
Dr, Birkmayer suggested that higher doses of NADH would be even more effective, e.g 40mg or higher dose of NADH.